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Gastric ulcer

Gastric ulcer - is a chronic disease in which the result of various disorders in the stomach or duodenum ulcer is formed (rarely two tetracycline antibiotics or more ulcers).

Peptic ulcer disease is associated with impaired mechanisms regulating the secretory, motor functions of the stomach and duodenum, the blood circulation in them, the power of the mucous membranes. Ulceration in the stomach or duodenum is only a consequence of the aforementioned disorders functions. Negative emotions, prolonged mental overstrain, pathological impulses from the affected internal organs in chronic appendicitis, chronic cholecystitis, cholelithiasis, etc. Are often the cause of peptic bodezni. Matter hormonal disorders. A role played by genetic factors (hereditary predisposition common among patients with peptic ulcer in 15-40 ° 6 cases). The immediate formation of an ulcer is the result of an imbalance between the "aggressive" (gastric juice, throw bile) and "protective" factors (gastric and duodenal mucus, cellular regeneration, normal local blood flow, the protective effect of certain hormones, as well as saliva alkaline reaction and pancreatic juice).

In the formation of ulcers in the stomach of the most important is a reduction of protective properties of the mucous membrane, weakening its resistance to the damaging effects of acid gastric juice. In the mechanism of the development of ulcers in the stomach and the output particularly in the duodenum, in contrast, the decisive factor is increased aggressiveness of gastric juice. Contributing factors are malnutrition regime, severe abuse, rough, irritating foods, constantly fast, hasty eating, drinking alcoholic beverages and their substitutes, smoking.

Pathogenesis (what's going on?) During Stomach ulcers:

The concept of the pathogenesis of peptic ulcer disease varied depending on the prevailing views in particular periods. Most of the theories of pathogenesis cause lang-voobrazovaniya tried to explain to some one position: inflammation, mechanical damage, vascular disorders, acid-peptic impact of impaired slizeobrazova-tion, back diffusion of H +, duodenal bile reflux, etc...

Numerous clinical and experimental studies in recent years greatly expanded our understanding of the local and neurohormonal mechanisms ulcerogenesis. From a modern point of view, the pathogenesis of peptic ulcer disease appears as a result of imbalance between the factors of gastric juice aggression and protect the mucous membrane of the stomach and duodenum.

Symptoms of stomach ulcers:

The most typical manifestation of a peptic ulcer is pain in the upper abdomen ( "half a spoon"), which occurs more often on an empty stomach, ie, between meals. Pain may also occur at night, forcing the patient to wake up and take food or medicine (which either inhibit the secretion of hydrochloric acid in the stomach, or neutralize it - the so-called antacids). The pain usually subsides within the first 30 minutes after a meal or taking these drugs.

A less specific, but occurring at a peptic ulcer symptoms include nausea, heaviness after eating, feeling of fullness, rarely bringing relief to vomiting, loss of appetite, weight gain, heartburn.

Treatment of gastric ulcers:

During exacerbation of patients hospitalized. Meals patients during treatment is divided into three stages.

During hospitalization, the first week of treatment for the disease is assigned a diet number 1a, the second week - a diet № 1b, hospital treatment in the last seven days is assigned to a diet number 1 .Before opening Helicobacter pylori treatment was based on antacid preparations, binders and envelop drugs and antispasmodics. Currently, the treatment of ulcers is carried out a course of antibiotics and anti-ulcer drugs.

Describe the specific treatment regimen is not feasible, as the treatment can be non-standard and depend on many factors such as the patient's age, his general physical condition, presence of concomitant diseases of the gastrointestinal tract (gastritis, duodenitis, and others). Gastric ulcer is quite a serious disease, which can result in perforation and death of the patient, so in any case, do not recommend you to self-medicate. Be sure to go through the diagnosis, consult a physician. And only then it is based on all laboratory data and assign other therapy.



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